http://publicationslist.org/data/diego.forero/atom.xml Diego Forerohttp://publicationslist.org/diego.forero$basepathfavicon.icoRecent additions to Diego Forero's PublicationsList.org pagehttp://publicationslist.org/publications.png2009-06-13T16:31:59Z http://publicationslist.org/diego.forero/refid5 2009-06-13T16:30:03Z

Through active reuptake of serotonin into presynaptic neurons, the serotonin transporter (5-HTT) plays an important role in regulating serotonin concentrations in the brain, and it is the site of binding for tricyclic antidepressants and selective serotonin reuptake inhibitors (SSRIs). Therefore it has been hypothesized that this transporter is involved in the etiology of bipolar (BP) disorder. In...<br/><br/>Maaike Alaerts, Shana Ceulemans, Diego Forero, Lotte N Moens, Sonia De Zutter, Lien Heyrman, An-Sofie Lenaerts, Karl-Fredrik Norrback, Dirk Goossens, Peter De Rijk, Lars-Göran Nilsson, Rolf Adolfsson, Jurgen Del-Favero (2009) <i>Am J Med Genet B Neuropsychiatr Genet</i> 150B: 4 585-592<br/>

http://publicationslist.org/diego.forero/refid1 2009-06-13T16:28:08Z

Analysis of genetic susceptibility factors for Alzheimer's disease (AD) in populations with different genetic and environmental background may be useful to understand AD etiology. There are few genetic association studies of AD in Latin America. In the present work, we analyzed polymorphisms in 3 candidate genes; the LDL receptor related protein-1, the microtubule-associated protein Tau and the se...<br/><br/>D A Forero, G Arboleda, J J Yunis, R Pardo, H Arboleda (2006) <i>J Neural Transm</i> 113: 9 1253-1262<br/>

http://publicationslist.org/diego.forero/refid2 2009-06-13T16:28:39Z

In recent years, it has been proposed that synaptic dysfunction may be an important etiological factor for Alzheimer's disease (AD). This hypothesis has important implications for the analysis of AD genetic risk in case-control studies. In the present work, we analyzed common functional polymorphisms in three synaptic plasticity-related genes (brain-derived neurotrophic factor, BDNF Val66Met; cate...<br/><br/>Diego A Forero, Bruno Benítez, Gonzalo Arboleda, Juan J Yunis, Rodrigo Pardo, Humberto Arboleda (2006) <i>Neurosci Res</i> 55: 3 334-341<br/>

http://publicationslist.org/diego.forero/refid3 2009-06-13T16:29:13Z

BACKGROUND: Genetic analysis of human longevity may be useful for the understanding of molecular mechanisms implicated in age-related diseases. The molecular genetics of human longevity is largely unexplored in Latin American populations and other developing countries. METHODS: To explore the possibility of an association of common polymorphisms in two candidate genes and longevity in Colombia, we...<br/><br/>Diego A Forero, Jorge Pinzón, Gonzalo H Arboleda, Juan J Yunis, Claudia Alvarez, Nohra Cataño, Humberto Arboleda (2006) <i>Arch Med Res</i> 37: 7 890-894<br/>

http://publicationslist.org/diego.forero/refid4 2009-06-13T16:29:43Z

In this paper, we review experimental advances in molecular neurobiology of Alzheimer's disease (AD), with special emphasis on analysis of neural function of proteins involved in AD pathogenesis, their relation with several signaling pathways and with oxidative stress in neurons. Molecular genetic studies have found that mutations in APP, PS1 and PS2 genes and polymorphisms in APOE gene are implic...<br/><br/>D A Forero, G Casadesus, G Perry, H Arboleda (2006) <i>J Cell Mol Med</i> 10: 3 796-805<br/>