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Hans Peter Müller

Institute of Clinical Dentistry, Faculty of Health Sciences, Tromsø University, 9037 Breivika, Norway

Phone:+47 776 49117
Fax: +47 776 49101
Mobile: +47 92050122
hans-peter.muller@uit.no
Professor of Periodontology, Institute of Clinical Dentistry, Faculty of Health Sciences, University of Tromsø, Norway, 2007

Vice Dean for Research and Student Affairs, Faculty of Dentistry, Kuwait University, 2006

Professor of Periodontology, Department of Surgical Sciences, Faculty of Dentistry, Kuwait University, 2005

Associate Professor, Department of Surgical Sciences, Faculty of Dentistry, Kuwait University, 2001

Professor of Dental, Oral, and Maxillofacial Medicine (apl), University of Heidelberg, 1999

Habilitation (PhD equivalent), Faculty of Medicine, University Münster, 1993

Specialist in Periodontology, Department of Periodontology, Faculty of Medicine, University of Münster, 1992

Dr. med. dent., Department of Periodontology, Faculty of Medicine, University of Marburg, 1981

Lic. Dent., Faculty of Medicine, University of Marburg, 1980

Languages (Reading, Writing, Speaking)
English: Fluent, Fluent, Fluent
German: Fluent, Fluent, Fluent
Norsk (Bokmål): Functional, Functional

Books

2012
H P Müller (2012)  Checkliste Parodontologie   Stuttgart: Georg Thieme Verlag 3rd ed.  
Abstract:
Notes: http://webshop.thieme.de/webshop/product/thieme/9783131263636/detail.jsf
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Journal articles

2013
E Könönen, H P Müller (2013)  Microbiology of aggressive periodontitis   Periodontology 2000, in press  
Abstract: For decades, Aggregatibacter actinomycetemcomitans has been considered the most likely etiologic agent in aggressive periodontitis. Implementation of DNA-based microbiologic methodologies has considerably improved our understanding of the composition of subgingival biofilms, and advanced open-ended molecular techniques even allow for genome mapping of the whole bacterial spectrum in a sample and characterization of both the cultivable and not-yet-cultivable microbiota associated with periodontal health and disease. Currently, A. actinomycetemcomitans is regarded as a minor component of the resident oral microbiota and as an opportunistic pathogen in some individuals. Its specific JP2 clone, however, shows properties of a true exogenous pathogen and has an important role in the development of aggressive periodontitis in certain populations. Still, limited data exist on the impact of other microbes specifically in aggressive periodontitis. Despite a wide heterogeneity of bacteria, especially in subgingival samples collected from patients, bacteria of the red complex in particular, and those of the orange complex, are considered as potential pathogens in generalized aggressive periodontitis. These types of bacterial findings closely resemble those found for chronic periodontitis, representing a mixed polymicrobial infection without a clear association with any specific microorganism. In aggressive periodontitis, the role of novel and not-yet-cultivable bacteria has not yet been elucidated. There are geographic and ethnic differences in the carriage of periodontitis-associated microorganisms, and they need to be taken into account when comparing study reports on periodontal microbiology in different study populations. In the present review, we provide an overview on the colonization of potential periodontal pathogens in childhood and adolescence, and on specific microorganisms that have been suspected for their role in the initiation and progression of aggressive forms of periodontal disease.
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Book chapters

2005
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Letter

2004
1999

Internet article

2003
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