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{userid:"karapanagioti", "articletype":"article","pages":"6516-6526","author":"S Ahn, D Werner, H K Karapanagioti, D R McGlothlin, R N Zare, R G Luthy","year":"2005","title":"Phenanthrene and pyrene sorption and intraparticle diffusion in polyoxymethylene, coke, and activated carbon","month":"SEP 1","journal":"ENVIRONMENTAL SCIENCE & TECHNOLOGY","publisher":"","volume":"39","number":"17","note":"","tags":"","booktitle":"","editor":"","abstract":"","address":"","school":"","issn":"0013-936X","doi":"10.1021\/es050113o","isi":"WOS:000231723800035","pubmed":"","key":"Ahn2005","howpublished":"","urllink":"","refid":92,"weight":92}
,

{userid:"dorian.arnold", "articletype":"inproceedings","pages":"1115-1122","author":"Joshua D Goehner, Taylor L Groves, Dorian C Arnold, Dong H Ahn, Gregory L Lee","year":"2013","title":"An Optimal Algorithm for Extreme Scale Job Launching","month":"","journal":"","publisher":"","volume":"","number":"","note":"Appeared in The 11th IEEE International Symposium on Parallel and Distributed Processing with Applications (ISPA-13), July 16-18, 2013, Melbourne, Australia. Published in the described proceedings.","tags":"SSL, System Services, Job Launch","booktitle":"12th IEEE International Conference on Trust, Security and Privacy in Computing and Communications (TrustCom)","editor":" ","abstract":"","address":"Melbourne, Australia","school":"","issn":"","doi":"","isi":"","pubmed":"","key":"Goehner2013Optimal","howpublished":"","urllink":"","refid":170,"weight":170}
,

{userid:"taylorlgroves", "refid":"3","repocollections":"","attachment":"","_thumb":"","articletype":"inproceedings","sectionheading":"","title":"An Optimal Algorithm for Extreme Scale Job Launching","year":"2013","author":"Joshua D Goehner, Taylor L Groves, Dorian C Arnold, Dong H Ahn, Gregory L Lee","booktitle":"Trust, Security and Privacy in Computing and Communications (TrustCom), 2013 12th IEEE International Conference on","editor":" ","pages":"1115-1122","organization":"","address":"","publisher":"","doi":"","pubmed":"","pdflink":"http:\/\/taylorgroves.com\/mwiki\/images\/9\/92\/ISPA-140.pdf","urllink":"http:\/\/ieeexplore.ieee.org\/xpls\/abs_all.jsp?arnumber=6680956","abstract":"","note":"","tags":"SSL, middleware, DOE","weight":3,"month":"","journal":"","volume":"","number":"","school":"","issn":"","isi":"","key":"goehner2013optimal","howpublished":""}
,

{userid:"suzanne.dahlberg", "articletype":"article","pages":"1476-1481","author":"D E Gerber, S E Dahlberg, A B Sandler, D H Ahn, J H Schiller, J R Brahmer, D H Johnson","year":"2013","title":"Baseline tumour measurements predict survival in advanced non-small cell lung cancer.","month":"Sep","journal":"British journal of cancer","publisher":"","volume":"109","number":"6","note":"","tags":"Aged,Aged, 80 and over,Antibodies, Monoclonal, Humanized,Antineoplastic Combined Chemotherapy Protocols,Bevacizumab,Carboplatin,Carcinoma, Non-Small-Cell Lung,Female,Humans,Lung Neoplasms,Male,Neoplasm Invasiveness,Neoplasm Staging,Paclitaxel,Predictive Value of Tests,Prognosis,Proportional Hazards Models,Survival Analysis,Treatment Outcome","booktitle":"","editor":"","abstract":"The association between tumour measurements and survival has been studied extensively in early-stage and locally advanced non-small cell lung cancer (NSCLC). We analysed these factors in patients with advanced NSCLC.","address":"","school":"","issn":"1532-1827","doi":"10.1038\/bjc.2013.472","isi":"","pubmed":"23942074","key":"Gerber2013","howpublished":"","urllink":"","refid":24,"weight":24}
,

{userid:"yha111", "articletype":"article","pages":"480-486","author":"J W Choi, D J Yoon, H W Lee, D P Han, Y H Ahn","year":"1995","title":"Antisense GLUT1 RNA suppresses the transforming phenotypes of NIH 3T3 cells transformed by N-Ras.","month":"Dec","journal":"Yonsei Med J","publisher":"","volume":"36","number":"6","note":"","tags":"3T3 Cells,Animals,Base Sequence,Cell Line, Transformed,Cell Transformation, Neoplastic,Genes, ras,Glucose Transporter Type 1,Humans,Mice,Molecular Sequence Data,Monosaccharide Transport Proteins,Phenotype,RNA, Antisense,Tumor Cells, Cultured","booktitle":"","editor":"","abstract":"An antisense approach was attempted to investigate the role of antisense GLUT1 RNA in suppressing tumor cell phenotypes using N-ras-transformed NIH 3T3 cells. The established cell line transformed by ras showed typical biological characteristics of cancer cells, such as increased glucose transport, GLUT1 mRNA contents, and the ability to form colonies on the soft agar. In this system, the plasmids (pMAM-GLUT1(rev)) which can transcribe the antisense GLUT1 RNA were transfected and the accompanying changes in the phenotypes of the ras-transformed cells were observed. The expression of antisense GLUT1 RNA by induction with dexamethasone reduced the glucose transport by 30% (1.97 +\/- 0.13 nmoles) after 4 min incubation when compared to the non-induction group of transformed cell (2.85 +\/- 0.19 nmoles). Also, the number of colonies sized over 50 microns on the soft agar was reduced significantly in the antisense RNA expressing group compared to non-induction group. These results suggest that the expression of antisense GLUT1 RNA reduced the glucose transport and transforming potential in soft agar possibly by hybridization with GLUT1 mRNA in N-ras-transformed NIH 3T3 cells.","address":"","school":"","issn":"0513-5796","doi":"","isi":"","pubmed":"8599249","key":"Choi1995","howpublished":"","urllink":"","refid":29}
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{userid:"frank.troxell", "refid":"112","repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"Multiplexed deep sequencing analysis of ALK kinase domain identifies resistance mutations in relapsed patients following crizotinib treatment","year":"2013","author":"D Huang, D W Kim, A Kotsakis, S Deng, P Lira, S N Ho, N V Lee, P Vizcarra, J Q Cao, J G Christensen, T M Kim, J M Sun, J S Ahn, M J Ahn, K Park, M Mao","journal":"Genomics","volume":"","number":"","pages":"","month":"","doi":"10.1016\/j.ygeno.2013.02.006","pubmed":"23434628","pdflink":"","urllink":"http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/23434628","abstract":"The recently approved ALK kinase inhibitor crizotinib has demonstrated successful treatment of metastatic and late stage ALK fusion positive non-small cell lung cancer (NSCLC). However, the median duration of clinical benefit is ~10-11months due to the emergence of multiple and simultaneous resistance mechanisms in these tumors. Mutations in the ALK kinase domain confer resistance to crizotinib in about one-third of these patients. We developed a multiplex deep sequencing method using semiconductor sequencing technology to quickly detect resistance mutations within the ALK kinase domain from tumor biopsies. By applying a base-pair specific error-weighted mutation calling algorithm (BASCA) that we developed for this assay, genomic DNA analysis from thirteen relapsed patients revealed three known crizotinib resistance mutations, C1156Y, L1196M and G1269A. Our assay demonstrates robust and sensitive detection of ALK kinase mutations in NSCLC tumor samples and aids in the elucidation of resistance mechanisms pertinent to the clinical setting.","note":"Huang, Donghui xD;Kim, Dong-Wan xD;Kotsakis, Athanasios xD;Deng, Shibing xD;Lira, Paul xD;Ho, Steffan N xD;Lee, Nathan V xD;Vizcarra, Pamela xD;Cao, Joan Q xD;Christensen, James G xD;Kim, Tae Min xD;Sun, Jong-Mu xD;Ahn, Jin Seok xD;Ahn, Myung-Ju xD;Park, Keunchil xD;Mao, Mao xD;ENG xD;2013\/02\/26 06:00 xD;Genomics. 2013 Feb 20. pii: S0888-7543(13)00034-7. doi: 10.1016\/j.ygeno.2013.02.006.","tags":"DNA Sequencing, Targeted DNA Sequencing, ALK kinase domain, Ion PGM, Torrent Suite v2.2, Ion 314 Chip, Cancer Research, PGM, analysis, crizotinib,deep sequencing,Mutation,resistance,sequencing, DNA Sequencing,Targeted DNA Sequencing,ALK kinase domain,Ion PGM,Torrent Suite v2.2,Ion 314 Chip,Cancer research","publisher":"","booktitle":"","editor":"","address":"","school":"","issn":"","isi":"","key":"Huang2013","howpublished":"http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/23434628"}
,

{userid:"yha111", "articletype":"article","pages":"117-125","author":"Y H Ahn, D J Yoon, G S Han, B G Lee","year":"1993","title":"Cloning and expression of rat liver type glucose transporter and translocation by insulin in Chinese hamster ovary cells.","month":"Jun","journal":"Yonsei Med J","publisher":"","volume":"34","number":"2","note":"","tags":"Animals,Base Sequence,CHO Cells,Cloning, Molecular,Cricetinae,Glucose Transporter Type 2,Insulin,Liver,Molecular Sequence Data,Monosaccharide Transport Proteins,Oligonucleotide Probes,Translocation, Genetic","booktitle":"","editor":"","abstract":"The 5'- and 3'-side half of liver type glucose transporter (GLUT2) cDNA was amplified from total RNA or mRNA by reverse transcriptase-polymerase chain reaction (RT-PCR). The amplified 5'-side fragment of GLUT2 cDNA was inserted into pGEM4Z and named pGLGT1, and the 3'-side fragment of GLUT2 cDNA was inserted into the HindIII site of pGLGT1 to construct pGLGT2 which contains an entire open reading frame of GLUT2 cDNA. The GLUT2 cDNA in pGLGT2 was transferred to an eukaryotic expression vector (pMAM) to construct pMLGT, which was expressed in the insulin-sensitive Chinese hamster ovary (CHO) cells. Western blot analysis showed that the GLUT2 gene in pMLGT was expressed in the transfected CHO cells successfully. The GLUT2 content in the plasma membrane fraction of insulin-treated CHO cells expressing GLUT2 increased 3.8-fold compared to that of the control group. This result suggests that GLUT2, which is not subjected to translocation by insulin in the cells of its major distribution, can be translocated if it is expressed in the suitable cells sensitive to insulin action.","address":"","school":"","issn":"0513-5796","doi":"","isi":"","pubmed":"8379190","key":"Ahn1993","howpublished":"","urllink":"","refid":31}
,

{userid:"yha111", "articletype":"article","pages":"1-16","author":"B L Daugherty, K Hotta, C Kumar, Y H Ahn, J D Zhu, S Pestka","year":"1989","title":"Antisense RNA: effect of ribosome binding sites, target location, size, and concentration on the translation of specific mRNA molecules.","month":"Jan\/Feb","journal":"Gene analysis techniques","publisher":"","volume":"6","number":"1","note":"","tags":"Binding Sites,Protein Biosynthesis,RNA,RNA, Antisense,RNA, Messenger,Ribosomes,beta-Galactosidase","booktitle":"","editor":"","abstract":"A series of plasmids were constructed to generate RNA complementary to the beta-galactosidase messenger RNA under control of the phage lambda PL promoter. These plasmids generate anti-lacZ mRNA bearing or lacking a synthetic ribosome binding site adjacent to the lambda PL promoter and\/or the lacZ ribosome binding site in reverse orientation. Fragments of lacZ DNA from the 5' and\/or the 3' region were used in these constructions. When these anti-mRNA molecules were produced in Escherichia coli 294, maximal inhibition of beta-galactosidase synthesis occurred when a functional ribosome binding site was present near the 5' end of the anti-mRNA and the anti-mRNA synthesized was complementary to the 5' region of the mRNA corresponding to the lacZ ribosome binding site and\/or the 5'-coding sequence. Anti-mRNAs producing maximal inhibition of beta-galactosidase synthesis exhibited an anti-lacZ mRNA:normal lacZ mRNA ratio of 100:1 or higher. Those showing lower levels of inhibition exhibited much lower anti-lacZ mRNA:normal lacZ mRNA ratios. A functional ribosome binding site at the 5'-end was found to decrease the decay rate of the anti-lacZ mRNAs. In addition, the incorporation of a transcription terminator just downstream of the antisense segment provided for more efficient inhibition of lacZ mRNA translation due to synthesis of smaller and more abundant anti-lacZ mRNAs. The optimal constructions produced undetectable levels of beta-galactosidase synthesis.","address":"","school":"","issn":"0735-0651","doi":"","isi":"","pubmed":"2472339","key":"Daugherty1989","howpublished":"","urllink":"","refid":53,"weight":53}
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{userid:"atulya.nagar", "articletype":"article","pages":"","author":"Ahamad Tajudin Khader, Andrei Paun, Atulya K Nagar, Azman Samsudin, Bahari Belaton, B S Bhattacharya, Chang Wook Ahn, Chee Peng Lim, D Sankar, D G Thomas","year":"","title":"Rajkumar Dare, Madras Christian College, Tambaram, Chennai, India Ronaldo Menezes, Florida Tech University Blvd, USA SR Thangiah, Slippery Rock University, PA, USA Shan He, University of Birmingham, United Kingdom Thamburaj Robinson, Madras Christian College, India","month":"","journal":"","publisher":"","volume":"","number":"","note":"","tags":"","booktitle":"","editor":"","abstract":"","address":"","school":"","issn":"","doi":"","isi":"","pubmed":"","key":"ref91","howpublished":"","urllink":"","refid":161,"weight":161}
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{userid:"atulya.nagar", "articletype":"article","pages":"","author":"Abdullah Zawawi Talib, Abdulqader Mohammed Abdulrahman Mohsen, Ahamad Tajudin Khader, Andrei Paun, Atulya K Nagar, B S Bhattacharya, Chang Wook Ahn, Chilukuri K Mohan, D G Thomas, Don Sofge","year":"","title":"Technical Programme Committee","month":"","journal":"","publisher":"","volume":"","number":"","note":"","tags":"","booktitle":"","editor":"","abstract":"","address":"","school":"","issn":"","doi":"","isi":"","pubmed":"","key":"ref108","howpublished":"","urllink":"","refid":178,"weight":178}
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