// +author:m arora +author:arora 
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{userid:"sugathan", "refid":86,"repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"Asymmetric fission around lead: The case of 198Po","year":"2019","author":"Shilpi Gupta, C Schmitt, K Mahata, A Shrivastava, P Sugathan, A Jhingan, KS Golda, N Saneesh, M Kumar, G Kaur, L Stuttg\u00e9, D Arora, H Arora, A Chatterjee, K Chauhan, SK Duggi, DP Kaur, V Mishra, Prashant N Patil, K Rani","journal":"Physical Review C","volume":"100","number":"","pages":"064608","month":"","doi":"","pubmed":"","pdflink":"","urllink":"","abstract":"","note":"","tags":"","weight":86}
,

{userid:"schirmer", "refid":"105","repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"Detection of vascularity in wrist tenosynovitis: power doppler ultrasound compared with contrast-enhanced grey-scale ultrasound.","year":"2010","author":"Andrea S Klauser, Magdalena Franz, Rohit Arora, Gudrun M Feuchtner, Johann Gruber, Michael Schirmer, Werner R Jaschke, Markus F Gabl","journal":"Arthritis Res Ther","volume":"12","number":"6","pages":"","month":"11","doi":"10.1186\/ar3185","pubmed":"21062450","pdflink":"http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC3046516\/pdf\/ar3185.pdf","urllink":"","abstract":"We sought to assess vascularity in wrist tenosynovitis by using power Doppler ultrasound (PDUS) and to compare detection of intra- and peritendinous vascularity with that of contrast-enhanced grey-scale ultrasound (CEUS).","note":"","tags":"Female,Humans,Male,Neovascularization, Pathologic,Tenosynovitis,Ultrasonography,Wrist Joint","publisher":"","booktitle":"","editor":"","address":"","school":"","issn":"1478-6362","isi":"","key":"Klauser2010","howpublished":""}
,

{userid:"ravikumar", "refid":"3","repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"The role of hydrostatic stress in determining the bandgap of InN epilayers","year":"2007","author":"Abdul Kadir, Tapas Ganguli, Ravi Kumar, M. R. Gokhale, A. P. Shah, Sandip Ghosh, B. M. Arora, and Arnab Bhattacharya ","journal":"Appl. Phys. Lett.","volume":"91","number":"","pages":"111913","month":"08","doi":"10.1063\/1.2784199","pubmed":"","pdflink":"","urllink":"","abstract":"This letter establishes a correlation between the internal stress in InN epilayers and their optical properties such as the measured absorption band edge and photoluminescence emission wavelength. By a careful evaluation of the lattice constants of InN epilayers grown on c-plane sapphire substrates under various conditions by metalorganic vapor phase epitaxy, the authors find that the films are under primarily hydrostatic strain. The corresponding stress results in a shift in the band edge to higher energy. The effect is significant and may be responsible for some of the variations in InN bandgap reported in the literature.","note":"","tags":""}
,

{userid:"j.e.fitzgerald", "articletype":"article","pages":"1156-1166","author":"M J Johnston, P Singh, P H Pucher, J E F Fitzgerald, R Aggarwal, S Arora, A Darzi","year":"2015","title":"Systematic review with meta-analysis of the impact of surgical fellowship training on patient outcomes.","month":"Sep","journal":"The British journal of surgery","publisher":"","volume":"102","number":"10","note":"","tags":"Clinical Competence,Education, Medical, Continuing,General Surgery,Humans,Internship and Residency,Patient Outcome Assessment,Surgical Procedures, Operative","booktitle":"","editor":"","abstract":"The number of surgeons entering fellowship training before independent practice is increasing. This may have a negative impact on surgeons in training. The impact of fellowship training on patient outcomes is not yet known. This review aimed to investigate the impact of fellowship training in surgery on patient outcomes.","address":"","school":"","issn":"1365-2168","doi":"10.1002\/bjs.9860","isi":"","pubmed":"26122269","key":"Johnston2015","howpublished":"","urllink":"","refid":37,"weight":37}
,

{userid:"sugathan", "refid":89,"repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"Measurement of neutron multiplicity to investigate the role of entrance channel parameters on the nuclear dissipation","year":"2019","author":"NK Rai, A Gandhi, Ajay Kumar, N Saneesh, M Kumar, G Kaur, A Parihari, D Arora, KS Golda, A Jhingan, P Sugathan, TK Ghosh, Jhilam Sadhukhan, BK Nayak, Nabendu K Deb, S Biswas, A Chakraborty","journal":"Physical Review C","volume":"100","number":"","pages":"014614","month":"","doi":"","pubmed":"","pdflink":"","urllink":"","abstract":"","note":"","tags":"","weight":89}
,

{userid:"digby.f.warner", "articletype":"article","pages":"","author":"Atica Moosa, Dirk A Lamprecht, Kriti Arora, Clifton E Barry, Helena I M Boshoff, Thomas R Ioerger, Adrie J C Steyn, Valerie Mizrahi, Digby F Warner","year":"2017","title":"Susceptibility of Mycobacterium tuberculosis Cytochrome  Oxidase Mutants to Compounds Targeting the Terminal Respiratory Oxidase, Cytochrome .","month":"10","journal":"Antimicrobial agents and chemotherapy","publisher":"","volume":"61","number":"10","note":"","tags":"Antitubercular Agents,Cytochromes c,Drug Discovery,Electron Transport Complex IV,Genome, Bacterial,Microbial Sensitivity Tests,Mycobacterium tuberculosis,Oxidative Phosphorylation,Oxygen,Oxygen Consumption,Sequence Deletion","booktitle":"","editor":"","abstract":"We deleted subunits I () and II () of the  cytochrome  menaquinol oxidase. The resulting \u0394 and \u0394 mutants were hypersusceptible to compounds targeting the mycobacterial  menaquinol-cytochrome  oxidoreductase and exhibited bioenergetic profiles indistinguishable from strains deficient in the ABC-type transporter, CydDC, predicted to be essential for cytochrome  assembly. These results confirm CydAB and CydDC as potential targets for drugs aimed at inhibiting a terminal respiratory oxidase implicated in pathogenesis.","address":"","school":"","issn":"1098-6596","doi":"10.1128\/AAC.01338-17","isi":"","pubmed":"28760899","key":"Moosa2017","howpublished":"","urllink":"","refid":5,"weight":5}
,

{userid:"schirmer", "refid":"253","repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"Raman spectroscopy for postmortem interval estimation of human skeletal remains: A scoping review.","year":"2023","author":"C Woess, Christian W Huck, J Badzoka, C Kappacher, R Arora, R A Lindtner, Philipp Zelger, M Schirmer, W Rabl, Johannes Pallua","journal":"Journal of biophotonics","volume":"16","number":"10","pages":"e202300189","month":"Oct","doi":"10.1002\/jbio.202300189","pubmed":"37494000","pdflink":"","urllink":"","abstract":"Estimating postmortem intervals (PMI) is crucial in forensic investigations, providing insights into criminal cases and determining the time of death. PMI estimation relies on expert experience and a combination of thanatological data and environmental factors but is prone to errors. The lack of reliable methods for assessing PMI in bones and soft tissues necessitates a better understanding of bone decomposition. Several research groups have shown promise in PMI estimation in skeletal remains but lack valid data for forensic cases. Current methods are costly, time-consuming, and unreliable for PMIs over 5\u2009years. Raman spectroscopy (RS) can potentially estimate PMI by studying chemical modifications in bones and teeth correlated with burial time. This review summarizes RS applications, highlighting its potential as an innovative, nondestructive, and fast technique for PMI estimation in forensic medicine.","note":"","tags":"Humans,Body Remains,Postmortem Changes,Spectrum Analysis, Raman,Bone and Bones,Burial","weight":253,"publisher":"","booktitle":"","editor":"","address":"","school":"","issn":"1864-0648","isi":"","key":"Woess2023","howpublished":""}
,

{userid:"sugathan", "refid":"85","repocollections":"","attachment":"","_thumb":"","articletype":"inproceedings","sectionheading":"","title":"Neutron Multiplicity Measurements for the Near Super-heavy Nucleus 260Rf","year":"2020","author":" Meenu Thakur, B.R. Behera, Ruchi Mahajan, N. Saneesh, Gurpreet Kaur, M. Kumar, A. Yadav, Neeraj Kumar, Kavita Rani, H. Arora, D. Kaur, S. Narang, Kavita, R. Kumar, P. Sugathan, A. Jhingan, K. S. Golda, A. Chatterjee, S. Mandal, A. Saxena, S. Kailas, and Santanu Pal","booktitle":"Proceedings of 13th International Conference on Nucleus-Nucleus Collisions","editor":"","pages":"010005","organization":"","address":"","publisher":"","doi":"https:\/\/doi.org\/10.7566\/JPSCP.32.010005","pubmed":"","pdflink":"","urllink":"","abstract":"","note":"","tags":"","weight":85,"journal":"JPS Conf. Proc.","volume":"32","number":"","month":""}
,

{userid:"thkolet", "articletype":"article","pages":"H1917-26","author":"U Siedlecka, M Arora, T Kolettis, G K Soppa, J Lee, M A Stagg, S E Harding, M H Yacoub, C M Terracciano","year":"2008","title":"Effects of clenbuterol on contractility and Ca2+ homeostasis of isolated rat ventricular myocytes","month":"","journal":"Am J Physiol Heart Circ Physiol","publisher":"","volume":"295","number":"5","note":"0363-6135 (Print) xD;0363-6135 (Linking) xD;Comparative Study xD;Journal Article xD;Research Support, Non-U.S. Gov't","tags":"Adrenergic beta-Agonists\/*pharmacology,Adrenergic beta-Antagonists\/pharmacology,Albuterol\/pharmacology,Animals,Calcium Channels, L-Type\/metabolism,Calcium Signaling\/*drug effects,Cells, Cultured,Clenbuterol\/*pharmacology,Disease Models, Animal,Dose-Response Relationship, Drug,Fenoterol\/pharmacology,GTP-Binding Protein alpha Subunits, Gi-Go\/genetics\/metabolism,Heart Failure\/*drug therapy\/metabolism\/physiopathology,Heart Ventricles\/drug effects\/metabolism,Homeostasis,Muscarinic Antagonists\/pharmacology,Myocardial Contraction\/*drug effects,Myocytes, Cardiac\/*drug effects\/metabolism,Rats,Sarcomeres\/drug effects\/metabolism,Transfection","booktitle":"","editor":"","abstract":"Clenbuterol, a compound classified as a beta2-adrenoceptor (AR) agonist, has been employed in combination with left ventricular assist devices (LVADs) to treat patients with severe heart failure. Previous studies have shown that chronic administration of clenbuterol affects cardiac excitation-contraction coupling. However, the acute effects of clenbuterol and the signaling pathway involved remain undefined. We investigated the acute effects of clenbuterol on isolated ventricular myocyte sarcomere shortening, Ca2+ transients, and L-type Ca2+ current and compared these effects to two other clinically used beta2-AR agonists: fenoterol and salbutamol. Clenbuterol (30 microM) produced a negative inotropic response, whereas fenoterol showed a positive inotropic response. Salbutamol had no significant effects. Clenbuterol reduced Ca2+ transient amplitude and L-type Ca2+ current. Selective beta1-AR blockade did not affect the action of clenbuterol on sarcomere shortening but significantly reduced contractility in the presence of fenoterol and salbutamol (P < 0.05). Incubation with 2 microg\/ml pertussis toxin significantly reduced the negative inotropic effects of 30 microM clenbuterol. In addition, overexpression of inhibitory G protein (Gi) by adenoviral transfection induced a stronger clenbuterol-mediated negative inotropic effect, suggesting the involvement of the Gi protein. We conclude that clenbuterol does not increase and, at high concentrations, significantly depresses contractility of isolated ventricular myocytes, an effect not seen with fenoterol or salbutamol. In its negative inotropism, clenbuterol predominantly acts through Gi, and the consequent downstream signaling pathways activation may explain the beneficial effects observed during chronic administration of clenbuterol in patients treated with LVADs.","address":"","school":"","issn":"","doi":"","isi":"","pubmed":"","key":"Siedlecka2008","howpublished":"http:\/\/www.ncbi.nlm.nih.gov\/entrez\/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=18775853  ","urllink":"http:\/\/www.ncbi.nlm.nih.gov\/entrez\/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=18775853  ","refid":5}
,

{userid:"andrew.deans", "refid":"35","repocollections":"","attachment":"","_thumb":"","articletype":"article","sectionheading":"","title":"FANCM limits ALT activity by restricting telomeric replication stress induced by deregulated BLM and R-loops.","year":"2019","author":"Bruno Silva, Richard Pentz, Ana Margarida Figueira, Rajika Arora, Yong Woo Lee, Charlotte Hodson, Harry Wischnewski, Andrew J Deans, Claus M Azzalin","journal":"Nature communications","volume":"10","number":"1","pages":"","month":"05","doi":"10.1038\/s41467-019-10179-z","pubmed":"31138795","pdflink":"https:\/\/www.nature.com\/articles\/s41467-019-10179-z.pdf","urllink":"https:\/\/www.nature.com\/articles\/s41467-019-10179-z","abstract":"Telomerase negative immortal cancer cells elongate telomeres through the Alternative Lengthening of Telomeres (ALT) pathway. While sustained telomeric replicative stress is required to maintain ALT, it might also lead to cell death when excessive. Here, we show that the ATPase\/translocase activity of FANCM keeps telomeric replicative stress in check specifically in ALT cells. When FANCM is depleted in ALT cells, telomeres become dysfunctional, and cells stop proliferating and die. FANCM depletion also increases ALT-associated marks and de novo synthesis of telomeric DNA. Depletion of the BLM helicase reduces the telomeric replication stress and cell proliferation defects induced by FANCM inactivation. Finally, FANCM unwinds telomeric R-loops in vitro and suppresses their accumulation in cells. Overexpression of RNaseH1 completely abolishes the replication stress remaining in cells codepleted for FANCM and BLM. Thus, FANCM allows controlled ALT activity and ALT cell proliferation by limiting the toxicity of uncontrolled BLM and telomeric R-loops.","note":"","tags":"Cell Death,Cell Line, Tumor,Cell Proliferation,DNA Helicases,DNA Replication,HEK293 Cells,HeLa Cells,Humans,RecQ Helicases,Ribonuclease H,Telomere,Telomere Homeostasis","weight":35,"publisher":"","booktitle":"","editor":"","address":"","school":"","issn":"2041-1723","isi":"","key":"Silva2019","howpublished":""}
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