// +author:r attewell +author:attewell var _ajax_res = { hits: 2, first: 0, results: [ {userid:"ranstam", "articletype":"article","pages":"545-551","author":"L Hagmar, T Bellander, V Englander, J Ranstam, R Attewell, S Skerfving","year":"1986","title":"Mortality and cancer morbidity among workers in a chemical factory.","month":"Dec","journal":"Scand J Work Environ Health","publisher":"","volume":"12","number":"6","note":"","tags":"Adult,Aged,Chemical Industry,Humans,Male,Middle Aged,Neoplasms,Occupational Diseases,Retrospective Studies,Sweden","booktitle":"","editor":"","abstract":"A retrospective cohort study was performed on a group of 664 male workers employed for at least one month during the period 1942-1979 in a chemical factory. Both established and suspected carcinogens had been handled in the plant, primarily piperazine, but also urethane, ethylene oxide, formaldehyde, and organic solvents. A significantly increased mortality, compared with the regional death rate, was observed in the cohort. The increase was mainly due to violent deaths and cardiovascular diseases. No rise in death rates was observed for asthma, bronchitis or emphysema, in spite of other evidence of a high risk of occupational asthma, due to exposure to piperazine. A statistically significant increase in cancer morbidity was observed for malignant lymphoma\/myelomatosis when an induction latency time of at least 10 years was used. Furthermore, an increase in bronchial cancer was noted, but it was statistically significant only when an induction-latency time of at least 15 years was used. A case-referent study within the cohort did not reveal any significant association between any specific chemical exposure and cancer morbidity.","address":"","school":"","issn":"0355-3140","doi":"","pubmed":"3823803","key":"Hagmar1986","howpublished":"","urllink":"","refid":143} , {userid:"ranstam", "articletype":"article","pages":"550-558","author":"L G Johansson, M P Albin, K M Jakobsson, H E Welinder, P J Ranstam, R G Attewell","year":"1987","title":"Ferruginous bodies and pulmonary fibrosis in dead low to moderately exposed asbestos cement workers: histological examination.","month":"Aug","journal":"Br J Ind Med","publisher":"","volume":"44","number":"8","note":"","tags":"Asbestos,Female,Humans,Lung,Male,Metalloproteins,Occupational Diseases,Pulmonary Fibrosis,Smoking,Time Factors","booktitle":"","editor":"","abstract":"Histological slides from the lungs of 89 dead asbestos cement workers have been examined with respect to ferruginous bodies and fibrosis. The results have been compared with individually matched controls with no known exposure to asbestos, and related to asbestos exposure, expressed as duration of exposure and cumulative asbestos dose, and smoking habits. The asbestos cement workers studied had been employed for on average 15 years, with a mean cumulative dose of 26 fibre-years per ml (f-y\/ml). Clear dose-response relations between exposure (duration of exposure and cumulative asbestos dose) and level of ferruginous bodies were found. An association was evident already at a low cumulative dose (1-10 f-y\/ml). Fibrosis was more common and more pronounced among the exposed workers than among controls. An association between ferruginous bodies and fibrosis was also found. Among the controls, but not among exposed workers, there was an association between smoking history and fibrosis.","address":"","school":"","issn":"0007-1072","doi":"","pubmed":"3651354","key":"Johansson1987","howpublished":"","urllink":"","refid":132} ] } ; ajaxResultsLoaded(_ajax_res);